Lately, I had been reading about countries who have stopped the Cov-19 Vaccines because of blood clots causing heart attacks and strokes. I remembered last year reading about blood clots forming in people who had the Virus and had formed antibodies, but soon developed life threatening clots.
Here are some excerpts from my article on Cytokine Storms:
I have read much about Cytokine Storms in my research on Covid-19. This horrific “overdrive” of the Immune system is responsible for most deaths from the Virus.
If that is not scary enough, many health care professionals have said that a fast-tracked vaccine will most likely cause a Cytokine storm in people.
What exactly is a Cytokine Storm?
This fatal condition happens in late stage Covid 19. The patient’s immune system emits an enormous number of “Cytokines” (proteins) into the blood stream and instead of the immune system fighting the Virus, the Cytokines cause severe inflammation throughout the body and attacks all organs. This causes multi- organ failure and the patient dies.
Cytokine storm in children
We have all been reading that very few children even get Covid, and if they do – it is very mild or asymptomatic. I read an alarming study about children and Covid which every parent needs to read:
What we know about the new Covid-19-linked illness in children
A rare, Kawasaki-like disease is striking kids who have coronavirus antibodies.Doctors in at least 20 US states have seen kids who have fallen sick with a rare inflammatory illness similar to a condition known as Kawasaki disease. Symptoms include fever and inflammation and can affect organs, including the heart. The majority of the children have tested positive for Covid-19 or antibodies to the virus, and three have died.
Doctors and scientists have been calling the syndrome in children “A Kawaski-like” syndrome, but it is a Cytokine Storm and can be fatal.
New York City alone has reported 147 cases of the pediatric inflammatory syndrome so far, with about a third of cases occurring in children younger than 5, and about half between the ages of 5 and 14.
Clusters of the illness have also popped up in other Covid-19 hot spots, including California, New Jersey, and Illinois. The potentially deadly syndrome is also affecting a small number of children in other countries, including the UK, Spain, and now Italy.
A peer-reviewed study out of Italy, published May 13 in The Lancet, details the cases of 10 children who presented with this condition during the Covid-19 outbreak there. Eight of the 10 children tested positive for the SARS-CoV-2 virus that causes Covid-19 in an antibody test.
“Our study provides the first clear evidence of a link between SARS-CoV-2 infection and this inflammatory condition, and we hope it will help doctors around the world as we try to get to grips with this unknown virus,” said Lorenzo D’Antiga, lead author of the study from the Hospital Papa Giovanni XXIII in Bergamo, Italy, in a statement.
Lauren Henderson, an attending physician at Boston Children’s Hospital and assistant professor of pediatrics at Harvard Medical School, called it, “a very important study.”
Read rest of article HERE
Blood Clots Are Another Dangerous COVID-19 Mystery
April 24, 2020 — Hooman Poor, MD, was tired of watching his patients die, and it looked like another was slipping away.
She was on a ventilator, but it wasn’t helping. Oxygen wasn’t getting to her organs. Acid was building in her blood. Her body was in shock. Her kidneys were failing, but he couldn’t put her on dialysis. She was too sick and probably wouldn’t survive it.
Poor, a pulmonologist and critical care specialist at Mount Sinai Hospital in New York City, was about to call her family to deliver the crushing news.
As he studied her lab results, he noticed that like many other COVID-19 patients, she had high levels of protein pieces called D-dimers, which are left over when the body breaks up blood clots. Her body was desperately trying, but failing, to clear blood clots, Poor believed.
A Medical Mystery
Around the world, doctors caring for COVID-19 patients have been trying to make sense of the same thing. When they draw blood from COVID patients, it clots in the tubes. When nurses insert catheters for kidney dialysis and IV lines to draw blood, the tubes quickly become clogged with clots.
“Patients are making clots all over the place,” says Adam Cuker, MD, a hematologist and associate professor of medicine at the Hospital of the University of Pennsylvania. “That’s making management of these patients very challenging.”
Types of Thrombosis
In addition to the well-known breathing problems, blood clots are a significant danger for COVID-19 patients. Clots are causing patients with COVID to have heart attacks and strokes; form strange rashes on their skin; and get red, swollen wounds that look like frostbite on their fingers and toes. On autopsy, the small vessels of the lungs and bowels, liver, and kidneys of COVID patients are choked with clots.
With his patient desperately ill, Poor suspected he had nothing to lose, so he punted.
“This is screaming blood clots. Why don’t we try tPA and see if it works?” he said.
He gave his patient a powerful clot-busting drug that’s normally used to treat strokes. It’s risky. If used improperly, it can cause uncontrolled bleeding, which can be deadly.
Within 30 minutes of getting the drug, his patient showed signs of improvement. Her carbon dioxide levels dropped, and other signs linked to shock seemed to improve. She lived for about another week, before ultimately dying. Poor has tried the drug on other COVID-19 patients, too. It hasn’t helped them survive, but he feels like it has shown him something about the disease.
“It did bring to light the possibility that blood clots are playing a bigger role in this disorder than we previously appreciated,” he says.
Lessons from the Dead
Sharon Fox, MD, PhD, agrees with him. She’s a pathologist at Louisiana State University Health in New Orleans and has been doing autopsies on patients who have died with COVID-19. She’s finished 20 cases so far, and they share something in common: They are riddled with blood clots in the smallest vessels of the body. Lungs seem to be especially hard-hit. There, clots appear to have cut off blood flow to the small air sacs where blood cells would be exchanging oxygen and carbon dioxide.
“There’s no ability for the blood to flow through and exchange oxygen like it should,” she says.
Fox says the pattern of damage is striking.
“I’ve never had a series of cases like this, where they all look the same, and all of the lungs have a similar pattern. There are types of vascular injury at autopsy that I haven’t seen before. I would say it’s new,” she says.
Dangerous blood clotting is always a risk for critically ill patients. That’s especially true for those who are immobilized and on mechanical ventilators, as patients with COVID-19 often are. But a recent French study, which compared 150 patients with COVID-related respiratory failure who were treated in intensive care units to 145 patients who had respiratory failure, but were not infected with the new coronavirus, found significantly higher rates of blood clotting in the COVID patients.
“We still need more controlled data, but based on clinical observations and the few studies that have been published, it looks like thrombosis [blood clotting] is more common in these patients,” Cuker says.
Why blood congeals the way it does in some COVID patients is still an open question.
One theory is that the body launches an immune attack called a cytokine storm to fight the virus that becomes self-directed, causing cells to kill themselves in an attempt to shut down the infection. Doctors believe that for some patients, the immune attack can end up doing the body more damage than the virus itself. This hyper-inflamed state is itself a well-known risk for blood clots. Cytokine storms can cause a condition called disseminated intravascular coagulation, or DIC, where patients both bleed uncontrollably and clot too much at the same time.
Another possibility is that the virus may more directly cause the clotting.
New Insights into ‘Why’
A research letter published this week in The Lancet reported evidence of viral bodies of the new coronavirus invading endothelial cells. The endothelium is the lining of our blood vessels. It directs important functions of the vascular system like clotting and swelling.
Images captured with an electron microscope found traces of the coronavirus in endothelial cells in the heart, kidney, small bowel, and lung — pretty much all over the body. Researchers collected the tissues during autopsies of three patients who died of COVID-19.
How Do Your Lungs Work?
We take them for granted, but your lungs do an amazing job of taking in oxygen.
Study co-author Mandeep Mehra, MD, medical director of the Brigham and Women’s Heart and Vascular Center in Boston, says the findings suggest that the virus can directly infect the endothelium. He says that while COVID-19 can certainly cause breathing problems, he doesn’t think it’s just a lung disease.
“This is actually a disease of the endothelium,” he says.
Mehra says the infection starts in the lungs because breathing is the easiest way for the virus to enter the body. Once it infects the lung cells and begins to destroy them, it travels into the bloodstream. There, it infects endothelial cells, causing endotheliitis.
Mehra thinks this endotheliitis comes from not only the direct infection of the blood vessel cells, but also from the haywire cytokine storm that the body launches to fight it off. “We’ve shown evidence of both.”
He says this theory of infection explains some things that doctors have been trying to puzzle out.
For example, certain conditions like high blood pressure, diabetes, and heart disease stress the endothelium. It’s no surprise, then, that people who have these conditions are also the ones who get the sickest when they catch COVID-19.
It also helps to explain why patients have such low oxygen in their blood, but their lungs may not be as stiff as they typically are in patients who have respiratory distress with pneumonia.
Mehra explains that one consequence of endotheliitis is that blood vessels can’t constrict the way they normally would. Typically, when a part of the lung becomes damaged, tiny blood vessels in that area close off so that blood will flow to a part of the lung that’s still working, where it can collect oxygen. This system protects the body from a sudden drop in oxygen, and it appears to break down in patients with severe COVID-19 infections. Mehra believes the infection of the endothelium is to blame.
The bottom line, he says, is that clotting is a feature of the COVID-19 syndrome. When it becomes a big problem, the disease is advanced and very severe. For that reason, treating the resulting blood clots probably won’t work.
He believes something worth trying might be to give patients drugs to support the endothelium, like ACE inhibitors and statins, along with anti-inflammatory drugs to tackle the cytokine storm, early in the course of the disease, but more research is needed to know for sure.
If COVID-19 is really an endothelial infection, Mehra thinks that also helps to explain why ventilators aren’t helping more patients. A study of 5,700 patients hospitalized with COVID-19 in New York City found that while just 12% needed a ventilator to help them breathe, 88% of those patients died.
“It’s not acting like influenza or other bacteria pneumonia where you get inflammation in the lungs and fluid buildup and very stiff lungs. That doesn’t seem to be the case, at least early on,” Poor says.
“The virus is acting as if its primary target is the endothelium,” he says.
That means the clots are just part of a much larger problem, one Poor says doctors don’t yet know how to solve. Source
Dr. Mercola interviewed Robert F. Kennedy Jr. on the Vaccine:
“Coronavirus Vaccine May Be a Disaster Waiting to Happen”
Kennedy goes on to summarize the history of coronavirus vaccine development, which began after three SARS epidemics had broken out, starting in early 2002.
“The first one was a natural epidemic that had moved from bats to human beings. The second two were lab-created organisms where people were experimenting with the coronavirus … That’s noncontroversial. Everybody accepts that.
“The Chinese, the Americans, the Europeans all got together and said, ‘We need to develop a vaccine against coronavirus.’ Around 2012, they had about 30 vaccines that looked promising. They took the four best of those and … manufactured the vaccines. They gave those vaccines to ferrets, which are the closest analogy when you’re looking at lung infections in human beings.
“The ferrets had an extraordinarily good antibody response, and that is the metric by which FDA licenses vaccines. Vaccines, as you know, are never tested in the field. They never give 5,000 people the vaccine, 5,000 people a placebo vaccine, and then tell them to go out and live life and watch what happens to those people. That never happens.
“The way that vaccines get licensed is that FDA gives people a vaccine or the industry gives them the vaccines, and then they do a serological response [test to] see ‘Did you develop in your blood antibodies to that target virus?’ The ferrets developed very strong antibodies, so they thought, ‘We hit the jackpot.’ All four of these vaccines … worked like a charm.
“Then something terrible happened. Those ferrets were then exposed to the wild virus, and they all died. [They developed] inflammation (cytokine storm) in all their organs, their lungs stopped functioning and they died.
“Then those scientists remembered that the same thing had happened in the 1960s when they tried to develop an RSV vaccine, which is an upper respiratory illness very similar to coronavirus.
“At the time, they did not test it on animals. They went right to human testing. They tested it on I think about 35 children, and the same thing happened. The children developed a champion antibody response, robust, durable. It looked perfect, and then the children were exposed to the wild virus and they all became sick. Two of them died. They abandoned the vaccine. It was a big embarrassment to FDA and NIH …
“Those scientists in 2012 remembered that, and they said, ‘This is the same thing that happened [back then].’ So, they look closer and they realize that there are two kinds of antibodies that were being produced by the coronavirus. There are neutralizing antibodies, which are the kind you want, which fight the disease, and then there are binding antibodies.
The binding antibodies actually create a pathway for the disease in your body, and they trigger something called … a paradoxical immune response or paradoxical immune enhancement. What that means is that it looks good until you get the disease, and then it makes the disease much, much worse …
“Coronavirus vaccines can be very dangerous, and that’s why even our enemies, people who hate you and me — Peter Hotez, Paul Offit, Ian Lipkin — are all saying, ‘You got to be really, really careful with this vaccine.’” source
Academics speak of the success of HCQ for early stage Covid-19
Read Article HERE
The reason I am not posting the text of this article is that it is from Oxford University, and the language used is far above my head and probably most of the readerships’ heads.
But a physician who prescribed HCQ for me explained that HCQ gets into our cells; changes the pH in the cells to prevent Covid 19 from entering our cells. He told me that HCQ must be taken in early stage Covid 19. It will prevent a Cytokine storm from developing in our bodies.
I do hope that there are some doctors, nurses or scientists reading this article. They will certainly understand the verbiage used in the Oxford piece. Here is a graphic from the article about the efficacy of HCQ on early stage Covid 19.
Safety of Hydroxychloroquine:
This drug has been approved by the FDA for over 60 years. I have a friend who has a severe type of Rheumatoid Arthritis. She has taken two 200 mg HCQ every day for the last 17 years with no side effects!
I am praying for so many people right now. We have Covid in our extended family.
Reading God’s Word and praying to Him is what we need to do.